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Maternal-fetal medicine is a subspecialty of obstetrics that encompasses the intricate care of pregnant individuals facing complex medical conditions or potential complications during pregnancy. Maternal-fetal medicine physicians, also known as perinatologists, are board-certified obstetricians with additional, specialized training in managing high-risk pregnancies.
Faculty
Faculty

Rochanda Mitchell, DO, RD (She/Her/Hers)

Faculty Physician

rochanda.mitchell@howard.edu

Department/Office

  • Centers of Excellence, Women's Health

School/College

  • College of Medicine

Education & Expertise

Education

Art

Bachelor
Fisk University
2004

Science

Master
Case Western Reserve
2006

Medicine of Osteopathy

Medical
Philadelphia College of Osteopathic Medicine
2013

Obstetrics and Gynecology Residency


Howard University College of Medicine
2018

Maternal Fetal Medicine Fellowship


University of Virginia
2021

Expertise

Maternal Fetal Medicine

Maternal-fetal medicine is a subspecialty of obstetrics encompassing the intricate care of pregnant individuals facing complex medical conditions or potential complications during pregnancy. Maternal-fetal medicine physicians, also known as perinatologists, are board-certified obstetricians with additional, specialized training in managing high-risk pregnancies.

Key areas of expertise in maternal-fetal medicine include:

Prenatal Diagnosis and Genetic Counseling: Proficient in utilizing advanced diagnostic technologies to detect and evaluate congenital anomalies, genetic disorders, and chromosomal abnormalities in the developing fetus. Provides expert genetic counseling to guide parents through decision-making processes.

High-Risk Pregnancy Management: Expertise in identifying and managing a wide spectrum of maternal medical conditions, such as diabetes, hypertension, autoimmune diseases, and infections, that may impact the pregnancy. Ensures optimal care to mitigate risks and complications.

Ultrasound and Imaging: Proficient in conducting specialized ultrasound examinations and fetal monitoring, allowing for early detection of fetal anomalies and growth restrictions.

Fetal Therapy: Knowledge in performing in-utero interventions when necessary, including fetal surgeries, transfusions, and medication administration to address specific fetal conditions.

Collaborative Care: Collaborates closely with a multidisciplinary team of healthcare professionals, including neonatologists, pediatric surgeons, and genetic counselors, to provide comprehensive care for the mother and the unborn child.

Preconception Counseling: Offers guidance to individuals with preexisting medical conditions or a history of complicated pregnancies to optimize health before conceiving.

Labor and Delivery Planning: Works with obstetricians to develop comprehensive labor and delivery plans tailored to the unique needs of high-risk pregnancies.

Patient Education and Support: Empowers expectant parents with information and emotional support, enabling them to make informed decisions about their pregnancy and the well-being of their child.

Related Articles

Impact of timing of multimodal analgesia in enhanced recovery after cesarean delivery protocols on postoperative opioids: A single center before-and-after study

Study objective Enhanced recovery after cesarean delivery (ERAC) programs aim to decrease maternal morbidity and aid in maternal recovery and return to baseline. Multimodal analgesia is an important element of ERAC protocols, but no consensus exists on the timing of medication administration. We compared maternal pain outcomes following scheduled cesarean delivery with modification of the timing of administration of multimodal analgesia with non-steroidal anti-inflammatory drugs (NSAIDs) and acetaminophen. Design Before-and-after study. Setting Labor and delivery unit at a single academic institution. Intervention NSAIDs and acetaminophen were administered as a fixed-interval alternating regimen every 3 h for the initial ERAC group (ERAC 1) and fixed-interval combined regimen every 6 h for the modified ERAC group (ERAC 2). ERAC 1 and ERAC 2 groups were compared to historical controls (Pre-ERAC). Patients 520 women undergoing scheduled cesarean delivery (Pre-ERAC n = 179, ERAC 1 n = 179, and ERAC 2 n = 162). Measurements The primary outcomes were postoperative total and daily opioid utilization as measured in morphine milligram equivalents (MME). Secondary outcomes included postoperative length of stay, maximum pain scores, and racial disparities in care. Main results The modified schedule of non-opioid analgesics involving combined administration (ERAC 2) versus alternating administration (ERAC 1) of multimodal analgesia resulted in decreased total postoperative opioid utilization (median = 26.3 vs 52.5 MME, Bonferroni corrected P = 0.002). Total postoperative opioid utilization among the ERAC 2 group was also significantly reduced compared to the Pre-ERAC group (median = 26.3 vs 105.0 MME, Bonferroni corrected P < 0.0001). Conclusions Multidisciplinary teams developing or modifying ERAC protocols for scheduled cesarean delivery should consider a combined administration at fixed intervals of NSAIDs and acetaminophen throughout the hospital stay to optimize postoperative pain management.

Study objective Enhanced recovery after cesarean delivery (ERAC) programs aim to decrease maternal morbidity and aid in maternal recovery and return to baseline. Multimodal analgesia is an important element of ERAC protocols, but no consensus exists on th

 

Diabetic neuropathy is manifested either by loss of nociception (painless syndrome) or by mechanical hyperalgesia and tactile allodynia (pain in response to nonpainful stimuli). While therapies with vasodilators or neurotrophins reverse some functional and metabolic abnormalities in diabetic nerves, they only partially ameliorate neuropathic pain. The reported link between nociception and targets of the anti-inflammatory drug sulfasalazine prompted us to investigate its effect on neuropathic pain in diabetes. We examined the effects of sulfasalazine, salicylates, and the poly(ADP-ribose) polymerase-1 inhibitor PJ34 on altered nociception in streptozotocin-induced diabetic rats. We also evaluated the levels of sulfasalazine targets in sciatic nerves and dorsal root ganglia (DRG) of treated animals. Finally, we analyzed the development of tactile allodynia in diabetic mice lacking expression of the sulfasalazine target nuclear factor-kappaB (NF-kappaB) p50. Sulfasalazine completely blocked the development of tactile allodynia in diabetic rats, whereas relatively minor effects were observed with other salicylates and PJ34. Along with the behavioral findings, sciatic nerves and DRG from sulfasalazine-treated diabetic rats displayed a decrease in NF-kappaB p50 expression compared with untreated diabetic animals. Importantly, the absence of tactile allodynia in diabetic NF-kappaB p50(-/-) mice supported a role for NF-kappaB in diabetic neuropathy. Sulfasalazine treatment also increased inosine levels in sciatic nerves of diabetic rats. The complete inhibition of tactile allodynia in experimental diabetes by sulfasalazine may stem from its ability to regulate both NF-kappaB and inosine. Sulfasalazine might be useful in the treatment of nociceptive alterations in diabetic patients.